This Causes Type 2 Diabetes Not Just Glucose or Fats
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Top Comments (10)
Excellent presentation. To me, this is one of the most important health-related topics that we can discuss. Controlling insulin levels (preventing hyperinsulinemia) is the key to preventing all of the most common modern diseases -- heart disease, cancer, diabetes, etc. *EDIT:* I was intending to type more but it was time for me to go read some bed-time stories to my son. There were two major highlights for me: 1) It is mostly common knowledge that consuming carbohydrates (glucose/fructose) will elevate insulin levels, but it also seems to be true that *non-glucose factors can also trigger insulin production:* ➖ Artificial sweeteners ➖ Iron consumption ➖ Reactive oxygen species 2) Mitochondrial damage (via ROS) can contribute to diabetes Here is the paragraph that stood out to me: _"Reactive oxygen species (ROS) are chemically reactive molecules containing oxygen. In high amounts ROS cause damage known as oxidative stress. However, ROS form as a natural byproduct of metabolism, and modest ROS production has important roles in cell signaling. Conditions that increase redox, as we found with stimulatory glucose and MOG, can lead to production of modest amounts of ROS in mitochondria. Thus, redox and ROS are candidate signals for basal insulin secretion, and we asked whether the putative signal was essential or sufficient."_ So, we see that mitochondria can be exposed to oxidative stress. Sources include: carb consumption (glucose), ROS, MOG, etc. This oxidative stress can damage mitochondrial function and contribute to type 2 diabetes. "_impaired mitochondrial function is a cause of insulin insensitivity in myocytes and adipocytes as a result of insufficient supply of energy or defects in the insulin signaling pathway._ _...inhibition of respiration and oxidative phosphorylation by respiratory inhibitors or knockdown of genes involved in mitochondrial biogenesis can impair the differentiation of preadipocytes and response of adipocytes to insulin._ _...defective mitochondria also cause a decrease in adiponectin secretion that leads to decline glucose utilization of other tissues._ _...Taken together, we suggest that mitochondrial dysfunction plays a role in the pathophysiology of insulin insensitivity._ *Source:* _Mitochondrial dysfunction in insulin insensitivity: implication of mitochondrial role in type 2 diabetes_ _Chih-Hao Wang, Ching-Chu Wang, Yau-Huei Wei_ In other words, it isn't just elevated insulin that contributes to diabetes; Damaging mitochondria also contributes to diabetes. We've got to seriously cut down on our carbohydrate intake. And we need to cut out processed foods since they contain artificial sweeteners and ROS. It's making more and more sense to my why ketogenic diets -- diets that heavily restrict carbohydrate consumption are so effective at treating type II diabetes.
Processed salad dressings are a major source of emulsifiers.
It is time to put some light on this epidemic of insulin resistance. Thank you again Dr Been for the outstanding work you are doing
Thanks as always Doc. You are generous with your time and thorough in your presentations.
*You Did Not Choose To Be Classified As Diabetic, But You Can Choose To Fight Back Against Diabetes* 💪
9:00min processed food, prepared food, pre-packaged food Intermittent fasting, bariatic surgery
Just "WOW!" Thanks again, DrBeen!
I just ate a banana in North America. How many bananas would I have eaten here in 1800? I suspect none.
Electro-magnetic pollution may be contributing to ROS. I personally am addressing this angle as well as OMAD to keep insulin level low.
This is a researcher worth tracking long-term. How refreshing to see someone thinking outside the box and really evaluating accepted concepts and practices. I hope she is not closeted by big pharma. Keep up the good work Dr Been, in sharing and explaining everything! You are the best!
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Top Comments (10)
Excellent presentation. To me, this is one of the most important health-related topics that we can discuss. Controlling insulin levels (preventing hyperinsulinemia) is the key to preventing all of the most common modern diseases -- heart disease, cancer, diabetes, etc. *EDIT:* I was intending to type more but it was time for me to go read some bed-time stories to my son. There were two major highlights for me: 1) It is mostly common knowledge that consuming carbohydrates (glucose/fructose) will elevate insulin levels, but it also seems to be true that *non-glucose factors can also trigger insulin production:* ➖ Artificial sweeteners ➖ Iron consumption ➖ Reactive oxygen species 2) Mitochondrial damage (via ROS) can contribute to diabetes Here is the paragraph that stood out to me: _"Reactive oxygen species (ROS) are chemically reactive molecules containing oxygen. In high amounts ROS cause damage known as oxidative stress. However, ROS form as a natural byproduct of metabolism, and modest ROS production has important roles in cell signaling. Conditions that increase redox, as we found with stimulatory glucose and MOG, can lead to production of modest amounts of ROS in mitochondria. Thus, redox and ROS are candidate signals for basal insulin secretion, and we asked whether the putative signal was essential or sufficient."_ So, we see that mitochondria can be exposed to oxidative stress. Sources include: carb consumption (glucose), ROS, MOG, etc. This oxidative stress can damage mitochondrial function and contribute to type 2 diabetes. "_impaired mitochondrial function is a cause of insulin insensitivity in myocytes and adipocytes as a result of insufficient supply of energy or defects in the insulin signaling pathway._ _...inhibition of respiration and oxidative phosphorylation by respiratory inhibitors or knockdown of genes involved in mitochondrial biogenesis can impair the differentiation of preadipocytes and response of adipocytes to insulin._ _...defective mitochondria also cause a decrease in adiponectin secretion that leads to decline glucose utilization of other tissues._ _...Taken together, we suggest that mitochondrial dysfunction plays a role in the pathophysiology of insulin insensitivity._ *Source:* _Mitochondrial dysfunction in insulin insensitivity: implication of mitochondrial role in type 2 diabetes_ _Chih-Hao Wang, Ching-Chu Wang, Yau-Huei Wei_ In other words, it isn't just elevated insulin that contributes to diabetes; Damaging mitochondria also contributes to diabetes. We've got to seriously cut down on our carbohydrate intake. And we need to cut out processed foods since they contain artificial sweeteners and ROS. It's making more and more sense to my why ketogenic diets -- diets that heavily restrict carbohydrate consumption are so effective at treating type II diabetes.
Processed salad dressings are a major source of emulsifiers.
It is time to put some light on this epidemic of insulin resistance. Thank you again Dr Been for the outstanding work you are doing
Thanks as always Doc. You are generous with your time and thorough in your presentations.
*You Did Not Choose To Be Classified As Diabetic, But You Can Choose To Fight Back Against Diabetes* 💪
9:00min processed food, prepared food, pre-packaged food Intermittent fasting, bariatic surgery
Just "WOW!" Thanks again, DrBeen!
I just ate a banana in North America. How many bananas would I have eaten here in 1800? I suspect none.
Electro-magnetic pollution may be contributing to ROS. I personally am addressing this angle as well as OMAD to keep insulin level low.
This is a researcher worth tracking long-term. How refreshing to see someone thinking outside the box and really evaluating accepted concepts and practices. I hope she is not closeted by big pharma. Keep up the good work Dr Been, in sharing and explaining everything! You are the best!